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. 2019 May 27;20(1):567–572. doi: 10.3892/mmr.2019.10301

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Copyright: © Ding et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Wnt/β-catenin signalling-mediated CEP cellular premature senescence. (A) A β-catenin inhibitor was associated with the recovery of cell proliferative ability. (B) LiCl treatment induced an increase in senescent cells, as determined by SA-β-gal staining, whereas the β-catenin inhibitor decreased the number of SA-β-gal-positive cells. *P<0.05. CEP-NOM, cells cultured under normal conditions; CEP-CTR, cells treated with LiCl; CEP-INB, cells treated with LiCl and a β-catenin inhibitor; CEP, cartilage endplate; LiCl, lithium chloride; SA-β-gal, senescence-associated β-galactosidase.