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. 2019 May 29;18(1):435–442. doi: 10.3892/etm.2019.7629

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — LncRNA-HOTAIR silencing reduces the sensitivity of drug resistance in drug-resistant MCF-7 cells. DOXR-MCF-7 cells were established and transfected with siRNA-HOTAIR1. (A) MTT (*P<0.05 and **P<0.01 vs. the NC group. ^#P<0.05 vs. the control group) and (B) colony formation assays with (C) subsequent quantification were performed to assess cell proliferation. ***P<0.001 vs. the NC group; ^###P<0.001 vs. the control group. LncRNA, long non-coding RNA; HOTAIR, HOX transcript antisense RNA; DOXR, doxorubicin resistant; siRNA, small interfering RNA; NC, negative control.