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. 2019 May 29;18(1):435–442. doi: 10.3892/etm.2019.7629

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — LncRNA-HOTAIR silencing inhibits proliferation and promotes apoptosis by regulating apoptosis associated proteins. (A) The protein expression of (B) Bax, (C) caspase-3 and (D) Bcl-2 and were determined via western blotting. Values are expressed as the mean ± standard deviation of three independent experiments. *P<0.05 vs. the NC group and ^#P<0.05 vs. the control group. LncRNA, long non-coding RNA; HOTAIR, HOX transcript antisense RNA; Bax, Bcl-2 associated X; VCl-2, B-cell lymphoma 2; siRNA/siR, small interfering RNA; NC, negative control; Dox, doxorubicin.