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. 2019 Jun 14;13:584. doi: 10.3389/fnins.2019.00584

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Copyright © 2019 Huang, Li, Zhou, Zhang, Chen, Wang, Xia, Liang and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Localization and expression of COX5A and effect of AQP4 inhibition in SCC. (A) IHC staining determined the localization of COX5A (brown) in motoneuron mainly following AQP4-RNAi lentivirus transduction. Black arrows mark COX5A. The group of AQP4-RNAi-LV had more obvious COX5A-positive products in motoneurons than the vector groups at 3, 7, and 28 days after SCC. Moreover, at 7 days as well as 28 days, there was a significant loss of COX5A-positive products in both the vector and AQP4-RNAi-LV groups. (B) The area of neurons in the AQP4-RNAi group was smaller than the vector groups at 3, 7, and 28 days after SCC. ^∗denotes P < 0.05, compared with vector groups. (C) The cell numbers of COX5A expressed in the AQP4-RNAi groups and the vector groups are shown. All of the AQP4-RNAi groups were higher than vector groups. ^∗denotes P < 0.05, compared with Vector groups.