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. Author manuscript; available in PMC: 2019 Jun 21.
Published in final edited form as: Free Radic Biol Med. 2019 Feb 18;134:630–643. doi: 10.1016/j.freeradbiomed.2019.02.015

Fig. 6. Treatment with UNC0638 reduced oxidative stress and cell death in the retina and optic nerve following TBI.

Fig. 6

(A–B) The confocal analysis shows that CellROX positive cells were decreased in TBI-mice treated with UNC0638 compared to TBI-mice in both optic nerve (A) and RGC region (B). (C–D) The quantitative analysis shows that TBARS were decreased in TBI-mice treated with UNC0638 compared to TBI-mice in both retinas (C) and optic nerve (D) sections. (E–F) The confocal shows that caspase 3 positive cells were decreased in TBI-mice treated with UNC0638 compared to TBI-mice in both retina (E) and optic nerve (F) sections. Statistical significance was measured by one-way ANOVA with a Tukey-Kramer post-hoc correction, n = 5, *p < 0.05. All data are expressed as mean ± S.E.M.