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. 2019 Jun 24;10(7):499. doi: 10.1038/s41419-019-1694-y

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a–c Cell proliferation assays were conducted to determine the effect of CLRN1-AS1 overexpression or knockdown on cell proliferation. d–e JC-1 assay and caspase-3 activity test were separately performed to demonstrate the role of CLRN1-AS1 in regulating cell apoptosis. f The levels of autophagy-related proteins in indicated pituitary prolactinoma cell lines. g The ratio of autophagosome in two transfected PPA cells. ^**P < 0.01 versus control group, indicated data are statistically significant