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. Author manuscript; available in PMC: 2020 Jun 21.
Published in final edited form as: Neurosci Lett. 2019 Apr 1;704:36–44. doi: 10.1016/j.neulet.2019.03.050

Table 4.

Genetic studies of 5-HTRs in AD.

Author, date Subjects (N) Study type Target
symptom(s)
Association
Holmes, 2003[81] AD, late-onset (158) Association study of selected SNPs in 5-HTR2A/2C Depression 5-HTR2A T102C; 5-HTR2C G68C(C23S)
Flirski, 2011[82] # articles dependent on foci Systematic review BPSD 5-HTR2C G68C(C23S) 5-HTT LPR
Tang, 2017[83] 9 eligible studies (2,005) Meta-analysis to investigate 5HTR2A T102C polymorphism BPSD 5HTR2A T102C
Liu, 2001 [84] AD (145) Controls (104) Association study of 5-HTR6 polymorphism C267T Depression 5-HTR6 C267T
Tsang, 2003[85] AD, no anxiety (22) AD, anxiety (12)
Aged Controls (n=14)
Association study Antemortem anxiety 5-HTT LPR
Engelborghs,
2013[97]
Probable AD (249) Prospective, longitudinal study for candidate genes BPSD 5-HTT LPR (SLC6A4), TPH2
Ueki, 2007[100] Mild AD (200)
(160 reached moderate AD)
Association study BPSD 5-HTT LPR,
5-HTTVNTR
ApoE
Sukonick, 2001[101] AD+Aggression (58) AD+No Aggression (79) Case-control study Aggression 5-HTT LPR
Bevilacqua,
2010[105]
Impulsive Males, unrelated (96) Association study in Finnish founder population Impulsivity 5-HTR2B stop codon (5HTR2B Q20*)
(exon-centric sequencing)
Montalvo-Ortiz,
2018[106]
AAs (3,269)
EAs (2,546)
AAs, from GTP (89)
GWAS Cannabis-related
aggression
HTR2B*rs17440378
Hollingworth,
2012[136]
AD+Psychosis (1,299) AD+No Psychosis (735) Controls (5,659) Combined analysis of three GWASs Psychosis SLC2A9*rs6834555,
VSNL1*rs4038131
Sherva, 2014[137] AD, discovery sample (303)1
AD, replication sample (323)2
GWAS Cognitive decline SP0N1*rs110231391,
SP0N1*rs116063452

Abbreviations: MDD = Major Depressive Disorder; GWAS=Genome-wide association study; AAs=African-Americans; EAs=European-Americans; GTP=Grady Trauma Project; ApoE= Apolipoprotein