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. 2019 Jun 24;11:5725–5735. doi: 10.2147/CMAR.S210782

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© 2019 Shen et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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TRIM14 regulated the apoptosis of colorectal cancer (CRC) cells. (A) HT-29 and SW620 cells were infected with shTRIM14 or shNC lentivirus. (B) LoVo cells were infected with pLVX-TRIM14 or pLVX lentivirus. At 48 hrs after infection, flow cytometry analysis was used to analyze apoptotic rates of CRC cells. shNC or pLVX served as negative control. ***P<0.001 vs shNC or pLVX.

Abbreviation: shNC, lentiviral plasmid expressing control short hairpin RNA.