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. 2018 May 4;74(2):155–162. doi: 10.1093/gerona/gly112

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Published by Oxford University Press on behalf of The Gerontological Society of America 2018.

This work is written by (a) US Government employee(s) and is in the public domain in the US.

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Figure 4. — NQO1 partly mediates the beneficial effects of caloric restriction (CR) on mitochondria. Immunoblot and densitometric quantification of (A, B) oxidative stress markers [Nrf2, SOD2, 4-HNE, and protein carbonylation (PCO)] and (C, D) mitochondrial players (PGC1α, Tfam, Sirt3), in liver of littermates (LM) and NQO1-KO mice under ad libitum (AL) or 40% CR. AL feeding (LM and KO) are represented by filled symbols and CR feeding (LM and KO) by open symbols. Data are mean ± SEM. *p < .05 compared to AL counterpart; #p < .05 compared to LM counterpart.