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. Author manuscript; available in PMC: 2020 Jul 1.
Published in final edited form as: Stroke. 2019 Jun 4;50(7):1727–1733. doi: 10.1161/STROKEAHA.119.024843

Table 1.

Univariate comparisons between patients with and without cerebellar microbleeds.

Patient without
cerebellar CMBs

N=172
Patient with
cerebellar CMBs

N=135
p-value
Age, years (SD) 70.4 (12.8) 69.7 (12.6) 0.6
Sex (female), n (%) 90 (52.3) 84 (62.2) 0.1
Hypertension, n (%) 121 (70.3) 117 (86.7) <0.001
Diabetes, n (%) 36 (20.9) 35 (25.9) 0.3
Hypercholesterolemia, n (%) 82 (47.7) 56 (41.5) 0.3
Warfarin use, n (%) 19 (11.0) 28 (20.7) 0.03
Antiplatelets use, n (%) 77 (44.8) 57 (42.2) 0.7
Presence of supratentorial CMBs, n (%) 88 (51.2) 96 (71.1) <0.001
Lacunes presence, n (%) 53 (30.8) 58 (42.9) 0.03
Severe CSO EPVS > 20, n (%) 70 (40.7) 57 (42.2) 0.8
Severe BG EPVS > 20, n (%) 36 (20.9) 35 (25.9) 0.3
WMH volume mL, median (IQR) 15 (8.2–29.7) 24 (11–41.2) <0.001
cSS presence, n (%) 13 (7.6) 18 (13.3) 0.3
Diagnosis of Probable CAA*, n (%) 49 (28.5) 38 (28.1) 1.0
Diagnosis of Deep/Mixed ICH, n (%) 72 (41.9) 78 (57.8) 0.006

SD: standard deviation, CMBs: cerebral microbleeds, CSO: Centrum Semiovale, EPVS: Enlarged perivascular spaces, cSS: cortical superficial siderosis, SVD: small vessel disease, WMH: white matter hyperintensities, IQR: interquartile range, CAA: cerebral amyloid angiopathy.

*

Probable CAA according to the modified Boston criteria