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. 2019 Jun 12;8(6):bio040121. doi: 10.1242/bio.040121

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© 2019. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Fig. 6. — BMP9 was involved in HPS and neutralized BMP9 with ALK1-FC restrained the expression level of KLF6, ALK1, ENG and PCNA in PMVECs stimulated by CBDL serum. (A,B) Representative immunohistochemistry detected (A) BMP9 and (B) ENG in sham 3-week CBDL rat lung (n=6). (C) Representative immunoblotting and quantification of KLF6, ALK1, ENG and PCNA in PMVECs treated with or without ALK1-FC in the presence of CBDL serum or normal serum for 48 h (n=3). Values are expressed as the mean±s.d. *P<0.05 compared with the sham group; ^#P<0.05 compared with the CBDL group.