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. 2019 Jul 1;10(7):506. doi: 10.1038/s41419-019-1737-4

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a, b Gene network analysis using GeneMANIA. ITGAV is the key factor among 20 genes that are related with IL-32 (a). The relationships between IL-32 and ITGAV are shown based on known functional association networks (b). c Human normal lung or NSCLC tissue sections (Grade I–III) were processed and stained; immunohistochemistry analyses for the expression of ITGAV were performed. The figures represent the sample of each cancer grade. d Tumors from mice injected with CD133+ B16F10 cells, and then tumors were collected after 85 days. Tumor tissue sections were analyzed by immunohistochemistry for the detection of ITGAV in non-Tg and IL-32γ Tg mice (upper lane). Tumors from nude mice injected with control vector-CD133+ or IL-32γ-CD133+ A549 cells were collected after 70 days. Tumor tissue sections were analyzed by immunohistochemistry for the detection of ITGAV (lower lane)