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. 2019 Jul 1;10(7):508. doi: 10.1038/s41419-019-1748-1

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — TCGA data showed that ZEB1 (a) and CCL8 (b) overexpression correlated with poor prognosis in human cervical cancer (P = 0.0335, P = 0.0019, respectively). *P < 0.05 by log-rank test. c Illustrative model showing the proposed mechanism by which hypoxia-induced ZEB1 potentiates TAM infiltration through its promotion of the secretion of CCL8 to form a metastasis-favourable TME