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. 2015 Sep 23;35(38):13160–13170. doi: 10.1523/JNEUROSCI.3123-14.2015

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Copyright © 2015 the authors 0270-6474/15/3513160-11$15.00/0

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Figure 5. — NMDAR activation and retrograde NO signaling are necessary for DPI. A, Sample traces of IPSCs before and after repetitive depolarization in the presence of the NO synthase inhibitor l-NAME (200 μm; top) and summary data from each cell showing l-NAME completely blocks potentiation of GABA synapses following repetitive depolarization (bottom; n = 6). B, Sample traces of IPSCs before and after repetitive depolarization in the presence of the sGC inhibitor ODQ (10 μm; top) and summary data from each cell showing ODQ completely blocks potentiation of GABA synapses following repetitive depolarization (bottom; n = 6). C, Sample traces of IPSCs before and after repetitive depolarization in the presence of the NMDAR antagonist APV (50 μm; top) and summary data from each cell showing APV completely blocks potentiation of GABA synapses following repetitive depolarization (bottom; n = 7). Calibrations: 25 pA, 10 ms.