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Possible antifibrotic mechanism of exogenous hydrogen sulfide in diabetic cardiomyocytes. H2S, hydrogen sulfide; TGF-β1, transforming growth factor-β1; R, receptor; GSK-3β, glycogen synthase kinase-3β; APC, adenomatous polyposis coli; p-, phosphorylated; Smad3, SMAD family member 3; P, phosphate; TCF/LEF, T-cell factor/lymphoid enhancer factor; WISP-1, Wnt1-inducible signaling pathway protein-1; MMP, matrix metalloproteinase; TIMP, tissue inhibitor of metalloproteinase.
