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. 2014 Aug 27;34(35):11571–11582. doi: 10.1523/JNEUROSCI.0231-14.2014

Table 5.

Significantly changed signaling pathways in caudal NPCs from EAE animalsa

Ingenuity canonical pathways −log (B-H p value) Downregulated No change Upregulated No overlap with dataset
PTEN signaling 4.5E00 28/135 (21%) 0/135 (0%) 2/135 (1%) 105/135 (78%)
PI3K/AKT signaling 3.74E00 28/144 (19%) 0/144 (0%) 1/144 (1%) 115/144 (80%)
Axonal guidance signaling 2.82E00 54/440 (12%) 0/440 (0%) 8/440 (2%) 378/440 (86%)
Reelin signaling in neurons 2.81E00 16/82 (20%) 0/82 (0%) 4/82 (5%) 62/82 (76%)
Signaling by Rho family GTPases 2.72E00 37/252 (15%) 0/252 (0%) 4/252 (2%) 211/252 (84%)
Integrin signaling 2.72E00 32/207 (15%) 0/207 (0%) 4/207 (2%) 171/207 (83%)
FGF signaling 2.56E00 17/92 (18%) 0/92 (0%) 3/92 (3%) 72/92 (78%)
Actin cytoskeleton signaling 2.56E00 33/238 (14%) 0/238 (0%) 4/238 (2%) 201/238 (84%)
IGF-1 signaling 2.49E00 20/105 (19%) 0/105 (0%) 1/105 (1%) 84/105 (80%)
NGF signaling 2.38E00 22/118 (19%) 0/118 (0%) 0/118 (0%) 96/118 (81%)
Ephrin receptor signaling 2.32E00 27/200 (14%) 0/200 (0%) 4/200 (2%) 169/200 (85%)
CXCR4 signaling 2.27E00 25/167 (15%) 0/167 (0%) 2/167 (1%) 140/167 (84%)
JAK/Stat signaling 1.94E00 14/70 (20%) 0/70 (0%) 1/70 (1%) 55/70 (79%)
CNTF signaling 1.91E00 12/55 (22%) 0/55 (0%) 0/55 (0%) 43/55 (78%)

aTable shows the significantly EAE-regulated canonical pathway in caudal NPCs. The analysis was performed using IPA. The p value is corrected for multiple comparisons using the Benjamini–Hochberg correction. The remaining columns show the number of molecules belonging to a certain pathway that were downregulated (column 3), unchanged (column 4), or upregulated (column 5) in our dataset. The percentages that these molecules constitute from the total number of molecules known to be involved in a certain pathway are shown in parentheses. The last column shows the number and percentage of the remaining molecules not regulated in our dataset but involved in a certain pathway.