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. 2019 Jul 4;10(7):514. doi: 10.1038/s41419-019-1707-x

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a The sequences of wild-type and mutant SNHG14 or EZH2 as well as miR-186-5p predicted by starBase 2.0. b, c The interaction of SNHG14, miR-186-5 p and EZH2 mRNA was proved by RNA pull down and RIP assays. d Luciferase reporter assays explained that SNHG14 and EZH2 mRNA bond with miR-186-5p by a competitive manner. e–g The impact of SNHG14 on EPHA7 expression was verified by carrying out qRT-PCR and ChIP. ^*P < 0.05, ^**P < 0.01, ^***P < 0.001