(A) Genome browser of maternal Otx1, Vegt, and Foxh1 binding with Pol II
binding, mesendodermal zygotic TF binding (b-catenin, Foxa, Gsc, Otx2, and
Smad2/3), and enhancer marks (Ep300, H3K27ac, and H3K4me1) near the genes
foxa4, pnhd, and
admp.
(B) Zygotically active TF ChIP-seq signal of b-catenin, Foxa, Gsc, Otx2,
and Smad2/3 in OVF-CRMs and 1- to 2-TF CRMs during the gastrula stage.
(C) Heatmap of H3K4me3 promoter mark ChIP-seq signal in OVF-CRMs,
1–2 TF CRMs, and gene promoters as a positive control.
(D–F) Heatmap of enhancer marks H3K4me1 (D), Ep300 (E), and
H3K27ac (F) ChIP-seq signal in OVF-CRMs and 1-to 2-TF CRMs.
(G) Fold change of Pol II ChIP-qPCR signal at gastrula stage 10.5 from
animal caps ectopically expressing otx1 and
vegt mRNA assaying for association with OVF-CRMs, compared
to uninjected animal caps. Shown is a single representative biological
replicate. The error bars represent the variation from 3 technical
replicates.
(H) Fold change of Pol II ChIP-qPCR signal at stage 10.5 from vegetal
masses in Otx1, Vegt, and Foxh1 triple morpholino KD conditions assaying for
association with OVF-CRMs, compared to uninjected vegetal masses. Shown is a
single representative biological replicate. The error bars represent the
variation from 3 technical replicates.
(I) RT-qPCR assay for eRNA transcription from OVF-CRMs in uninjected,
and otx1 and vegt mRNA injected animal caps at
stage 10.5. Genomic DNA contamination is quantified using an RT-qPCR control
reaction with no reverse transcriptase (–RT). Shown is a single
representative biological replicate. The error bars represent the variation from
3 technical replicates.
The experiments for (G)–(I) were repeated at least twice.