Table. Patient and Infarct Characteristics.
| Characteristic | Patients (n = 73) |
|---|---|
| Age, mean (SD), y | 60.8 (9.9) |
| Male, No. (%) | 59 (80.8) |
| Body mass index, mean (SD)a | 27.3 (3.7) |
| Medical history, No. (%) | |
| Hypertension | 19 (26.0) |
| Type 2 diabetes | 5 (6.8) |
| Current smoker | 29 (39.7) |
| Family history of CAD | 27 (37.0) |
| Hypercholesterolemia | 11 (15.1) |
| Previous percutaneous intervention | 2 (2.7) |
| History of pulmonary disease | 5 (6.8) |
| Medication before pPCI, No. (%) | |
| Aspirin | 4 (5.5) |
| Lipid-lowering medication | 10 (13.7) |
| Angiotensin-converting enzyme inhibitor | 4 (5.5) |
| Angiotensin receptor blocker | 7 (9.6) |
| β-Blocker | 6 (8.2) |
| Calcium channel blocker | 6 (8.2) |
| Peak laboratory values, median (IQR) | |
| Creatine kinase level, U/L | 1086.0 (467.5-2240.5) |
| CK-MB level, μg/L | 99.2 (43.3-224.0) |
| Troponin level, μg/L | 2.01 (0.53-4.52) |
| Culprit arteries, No. (%) | |
| Left anterior descending | 24 (32.9) |
| Circumflex | 19 (26.0) |
| Right coronary | 30 (41.1) |
| Angiographic and stent characteristics, No. (%) | |
| TIMI flow grade before pPCI 0 | 44 (60.3) |
| 1 | 7 (9.6) |
| 2 | 15 (20.5) |
| 3 | 7 (9.6) |
| TIMI flow grade after pPCI 0 | 0 |
| 1 | 4 (5.5) |
| 2 | 11 (15.1) |
| 3 | 58 (79.5) |
| cTFC | 38 (23-59) |
| Nonculprit arteries, No. (%) | |
| Left anterior descending | 29 (39.7)b |
| Circumflex | 26 (35.6)c |
| Right coronary | 18 (24.7) |
| QCA diameter stenosis, No. (%) | 55.1 (13.3) |
Abbreviations: CAD, coronary artery disease; IQR, interquartile range; cTFC, corrected TIMI frame count; pPCI, primary percutaneous coronary intervention; QCA, quantitative coronary angiography; TIMI, thrombolysis in myocardial infarction.
SI conversion factors: To convert creatine kinase to microkatals per liter, multiply by 0.0167; to convert CK-MB and troponin to micrograms per liter, multiply by 1.0.
Calculated as weight in kilograms divided by height in meters squared.
One patient included with the left anterior descending artery as the culprit vessel and the first diagonal branch as the nonculprit vessel. The culprit location was in the midleft anterior descending artery distal to the first diagonal branch (marked as left anterior descending in the Table).
One patient included with the circumflex artery as the culprit vessel and the intermediate branch (marked as circumflex in the Table) as the nonculprit vessel. The culprit location was midcircumflex.