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. 2019 Jul 3;11:6043–6059. doi: 10.2147/CMAR.S197583

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© 2019 Chen et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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LINC01116 can bind to miR-136, which targets FN1. (A) Binding sites of miR-136 and FN1. (B) Binding sites of miR-136 and LINC01116. (C) Measurement of luciferase activity to verify the relationship of miR-136 and FN1. (D) Measurement of luciferase activity to verify the relationship of miR-136 and LINC01116. The data were measurement data and expressed as mean±standard deviation. This experiment was repeated 3 times to obtain the mean value. The t-test was used for comparison between two groups. *p<0.05 vs the LINC01116-wt + NC group; ^#p<0.05 vs the FN1-wt + NC group.

Abbreviations: FN1, fibronectin1; miR-136, microRNA-136; wt, wild type; NC, negative control.