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. 2019 Jul 2;10:628. doi: 10.3389/fgene.2019.00628

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Copyright © 2019 Yang, Song, Chen, Yuan, Chen, Ding, Guan, McGrath, Song, Tong and Wang

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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(A–C) Images of renal pathology (II:3) under light microscopy by periodic acid-silver methe-namine (PASM) (A and B) and periodic acid-schiff (PAS) staining (C). A non-uniform glomerular basement membrane (GBM) (arrow), 200×. (B) Tubular atrophy and interstitial fibrosis, 200×. (C) The infiltration of foam cells (arrow) in renal interstitium. (D–F) Representative electron micrograph of renal tissue. (D) Some segments of GBM were absent (arrow). (E) Irregular GBM thinning, thickening, and splitting. (F) Effacement of podocyte foot processes (arrow). (G–I) Immunofluorescence staining of Col4A3 (Ab111742, Abcam, USA) in renal tissue from a patient with minimal change disease (MCD) (G, left panel), the proband (II:3) (H, middle panel), and a male patient with XLAS (I, right panel), 400×.