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. 2019 Jul 10;19:677. doi: 10.1186/s12885-019-5867-y

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — Aging APC2-deficient mice develop adult GCTs. Tumours ranged in size from (a) small in situ tumours (arrow) to (b) small tumours of normal ovarian size, (c, d) small but macroscopically visible tumours or (e) a large macroscopic tumour (arrow). The tumours displayed varying histologic patterns such as (f) follicular, (g) nodular (arrows), (h) insular, (i) luteinized (arrow: luteinized area shown at 4X original magnification in inset, L: luteinized, NL: non-luteinized), (j) diffuse (arrow: Call-Exner body), and (k) cystic patterns. The tumours were (l) highly vascularized, (m) anaplastic, (n) mitotic (black arrow), and showed (o) Call-Exner bodies (black arrow) and coffee-bean nuclei (yellow arrow). Bars a-d = 500 μm, f-l = 100 μm, m-n = 50 μm, o = 20 μm