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. Author manuscript; available in PMC: 2019 Jul 10.
Published in final edited form as: Pancreatology. 2019 Feb 26;19(4):500–506. doi: 10.1016/j.pan.2019.02.009

Table 2.

Comparison of physician identification of smoking as a chronic pancreatitis risk factor based on smoking status and alcohol etiology in the NAPS2 studies

Self-reported
smoking status
Alcohol
etiology
NAPS2-CV or
NAPS2-AS Study
NAPS2 Original Study
N Physician
identified
smoking as a
CP risk factor
n (%)
N Physician
identified
smoking as a
CP risk factor
n (%)
Ever Smoked 508 410 (80.7) 382 173 (45.3)
Yes 315 283 (89.8) 222 121 (54.5)
No 193 127 (65.8) 160 52 (32.5)
Current Smoker 334 305 (91.3) 253 134 (53.0)
Yes 236 220 (93.2) 177 98 (55.4)
No 98 85 (86.7) 76 36 (47.4)
Past Smoker 174 105 (60.3) 129 39 (30.2)
Yes 79 63 (79.8) 45 23 (51.1)
No 95 42 (44.2) 84 16 (19.0)
≥1 PPD 229 190 (83.0) 213 106 (49.8)
Yes 142 126 (88.7) 127 74 (58.3)
No 87 64 (73.6) 86 32 (37.2)
≤ 1PPD 275 218 (79.3) 147 58 (39.5)
Yes 170 155 (91.2) 85 42 (49.4)
No 105 63 (60.0) 62 16 (25.8)

Alcohol etiology was based on physician assessment

All comparisons between NAPS2-CV or NAPS2-AS studies vs. NAPS2 original study were ≤0.001.