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. 2019 Jul 3;10:737. doi: 10.3389/fphar.2019.00737

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Copyright © 2019 Ma, Liu, Liu, Wang, Chang, Li, Li, Hou and Bai

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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PEI inhibited the EGFR signaling pathway, and CBPP alleviated pulmonary fibrosis. (A) BEAS-2B cells were pretreatment with PEI, then stimulated by 1 ng/ml EGF. The phosphorylation of AKT^S473 and GSK3β was measured by Western blot, and the relative intensity data of P-AKT^S473 and P-GSK3β to GAPDH are represented by the mean ± SD of three groups, *p < 0.05 and **p < 0.01. (B) Masson staining images (40-fold) of lung pathological sections and (C) statistics for the score of histological fibrosis, **p < 0.01 and ***p < 0.001 compared to the Mod. ^### p < 0.001 compared to the Con (n = 6).