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. 2012 Aug 29;32(35):12005–12017. doi: 10.1523/JNEUROSCI.0871-12.2012

Figure 3.

Figure 3.

Effects of prepairing intra-amygdala injections of ROCK inhibitor Y27632, actin polymerization inhibitor latrunculin A, or NMDA receptor antagonist d-AP5 on Arc/Arg3.1 protein levels in homogenates and synaptic membrane fractions of amygdala isolated from rats 1 h after CMW. A, Intra-amygdala injections of Y27632 blocked enhancement of Arc/Arg3.1 protein expression in the synaptic membrane fractions of amygdala (right), but had no effect on increase in Arc/Arg3.1 protein expression in the homogenates of amygdala (left). B, Intra-amygdala injections of latrunculin A attenuated Arc/Arg3.1 expression in the synaptic membrane fractions of amygdala (right), but had no effect on increase in Arc/Arg3.1 protein expression in the homogenates of amygdala (left). C, Intra-amygdala injections of the NMDA receptor antagonist d-AP5 blocked enhancement of Arc/Arg3.1 protein expression in both the homogenates (left) and the synaptic membrane fractions (right) of amygdala. Error bars represent mean ± SEM. *p < 0.05, **p < 0.01, ***p < 0.001, compared with the corresponding control groups, one-way ANOVA with Newman–Keuls post hoc test. Homo, Homogenate fraction; Con, control; Veh, vehicle, Latr, Latrunculin A.