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. 2019 Mar 15;13(4):430–439. doi: 10.5009/gnl18316

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Copyright © 2019 by The Korean Society of Gastroenterology, the Korean Society of Gastrointestinal Endoscopy, the Korean Society of Neurogastroenterology and Motility, Korean College of Helicobacter and Upper Gastrointestinal Research, Korean Association the Study of Intestinal Diseases, the Korean Association for the Study of the Liver, Korean Pancreatobiliary Association, and Korean Society of Gastrointestinal Cancer.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 2 — Effects of Bifidobacterium infantis on Foxp3. (A–E) Foxp3 was detected by immunohistochemistry in the control, dextran sodium sulfate (DSS), DSS+B9, DSS+B8, and DSS+B7 groups (original magnification, ×400). (F) Analysis of Foxp3 expression by average optical density according to (A–E). (G) Foxp3 expression in each group by Western blot. (H) Analysis of Foxp3 according to (G). Values given as the mean±SD (*p<0.01, compared to the control group; ^†p<0.05, compared to the DSS group; ^‡p<0.05, compared to the DSS+B9 group; ^§p<0.05, compared to the DSS+B8 group).