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. 2019 Jul 12;12:75. doi: 10.1186/s13045-019-0762-1

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Genomic landscape of Chinese LUSC and its correlations with TMB, PD-L1 expression, or immune cells infiltration. a The identified somatic mutations of all cases. b The identified copy number variations of all samples. c–p Association between frequent alterations including TP53 (c), KMT2C (d), NFE2L2 (e), KEAP1 (f), CDKN2A (j), PTEN (k), FBXW7 (l) and PIK3CA (m) mutations, FGFR1 (g), PIK3CA (h), EGFR (i), CCND1 (n) and SOX2 (p) amplification, loss of CDKN2A (o), and TMB. q–r Association between frequent somatic mutations and PD-L1 expression (q), CD8+ TIL expression (r). s–t Association between frequent copy number variations and PD-L1 expression (s), CD8+ TIL expression (t)