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. 2019 Jun 6;20(2):931–938. doi: 10.3892/mmr.2019.10358

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Effects of RNF10 on the inflammatory responses in carotid artery injury. (A) RNF10, (B) MCP-1 and TNF-α mRNA expression levels were analyzed with qPCR (n≥3). (C) Representative western blot of NF-κB protein. (D) Quantification of NF-κB protein level as determined by western blot analysis (n≥3). &P<0.05 vs. the corresponding control group; *P<0.05 vs. the corresponding DM+Sham group; #P<0.05 vs. the corresponding DM+BI+Ad-GFP group. DM, diabetes mellitus; RNF10, RING finger protein 10; shRNF10, RNF10 short hairpin RNA; BI, balloon injury; Ad, adenovirus; GFP, green fluorescent protein; qPCR, quantitative polymerase chain reaction; TNF-α, tumor necrosis factor α; MCP-1, monocyte chemotactic protein 1; NF-κB, nuclear factor-κ-gene binding.