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. 2019 Jun 25;20(2):1725–1735. doi: 10.3892/mmr.2019.10421

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Copyright: © Xu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — miR-153 regulates cell growth. (A) Overexpression of miR-153 caused changes in the proliferation rate of HT-22 cells, ***P<0.001. (B) Cell cycle distribution of HT-22 cells with rLV-miR (a) and rLV-miR-153 overexpression (b) was determined by flow cytometry. Significance was determined by comparing G1/G0, S and G2 phase cells with miR-153 overexpression to control cells with multiple unpaired t-tests (c). *P<0.05, **P<0.01. (C) Overexpression of miR-153 caused reduction in CDK12 mRNA in HT-22 cells. Significance was determined by comparing miR-153 overexpression to control cells with two-tailed paired t-test. Error bars represent standard deviation. *P<0.05. CDK, cyclin-dependent kinase.

Figure 4. — miR-153 regulates cell growth. (A) Overexpression of miR-153 caused changes in the proliferation rate of HT-22 cells, ***P<0.001. (B) Cell cycle distribution of HT-22 cells with rLV-miR (a) and rLV-miR-153 overexpression (b) was determined by flow cytometry. Significance was determined by comparing G1/G0, S and G2 phase cells with miR-153 overexpression to control cells with multiple unpaired t-tests (c). *P<0.05, **P<0.01. (C) Overexpression of miR-153 caused reduction in CDK12 mRNA in HT-22 cells. Significance was determined by comparing miR-153 overexpression to control cells with two-tailed paired t-test. Error bars represent standard deviation. *P<0.05. CDK, cyclin-dependent kinase.