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. 2019 Jun 10;20(2):1541–1550. doi: 10.3892/mmr.2019.10371

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Copyright: © Zhu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — H/R reduces H9C2 cardiomyocyte viability and contributes to cell injury. (A) Effects of H/R on H9C2 cell viability following 0, 4, 8 and 12 h of reoxygenation, as determined by a Cell Counting Kit-8 assay. Effects of H/R on the following markers of cardiomyocyte injury: (B) LDH, (C) MDA and (D) SOD, following 0, 4, 8 and 12 h of reoxygenation. Data are presented as the mean ± standard deviation (n=3). *P<0.05, **P<0.01 vs. 0 h. H/R, hypoxia/reoxygenation; LDH, lactate dehydrogenase; MDA, malondialdehyde; OD, optical density; SOD, superoxide dismutase.