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. 2019 Jul 1;13(7):e0007486. doi: 10.1371/journal.pntd.0007486

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© 2019 Ratnayake et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 3 — Scatter plots of the maximum biomarker concentrations reached within 24 h post-bite for patients with no acute kidney injury (No AKI; green), mild AKI (grade 1; blue), moderate AKI (grade 2; orange) and severe AKI (grade 3; red), for serum creatinine (sCr; Panel A), serum cystatin C (sCysC; Panel B), urinary clusterin (uClu; Panel C), urinary neutrophil gelatinase-associated lipocalin (uNGAL; Panel D), kidney injury molecule-1 (uKIM-1; Panel E),albumin (uAlb; Panel F),beta2-microglobulin (uβ2M; Panel G), urinary cystatin C (uCysC; Panel H), osteopontin(uOPN; Panel I) and trefoil factor-3 (uTFF3; Panel J).