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vlsE and Silent Cassettes on Linear Plasmid 28-1 of B. burgdorferi—Borrelia evasion mechanisms counter both innate and adaptive immune responses in order to establish persistent infection in the immunocompetent vertebrate host. Random segments of silent cassettes 2–16 are recombined at the vlsE expression site near the end of lp28-1 to produce surface proteins with a high degree of antigenic variation.
