Skip to main content
. 2019 Jun 11;6(2):54. doi: 10.3390/vetsci6020054

Figure 1.

Figure 1

Effect of long-chain fatty acids (LCFA; palmitic acid [C16], stearic acid [C18], trans10, cis12 conjugated linoleic acid [CLA], docosahexaenoic acid [DHA], and eicosapentaenoic acid [EPA]), peroxisome proliferator-activated receptor gamma (PPARγ) agonist (ROSI; rosiglitazone) and LCFA + PPARγ antagonist (GW; GW9662) on transcription of genes related to fatty acid synthesis and desaturation and triglycerides synthesis. Lower case letters and uppercase letters denote statistical differences (p < 0.05) between treatments without GW and treatments with GW, respectively. * denotes statistical difference (p < 0.05) between presence and absence of GW in the same LCFA treatment. For GW treatment alone, * denotes statistical difference (p < 0.05) with the PCTR.