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. 2019 Jun 11;6(2):54. doi: 10.3390/vetsci6020054

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© 2019 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Effect of LCFA (palmitic acid [C16], stearic acid [C18], trans10, cis12 conjugated linoleic acid [CLA], docosahexaenoic acid [DHA], and eicosapentaenoic acid [EPA]), PPARγ agonist (ROSI; rosiglitazone) and LCFA + PPARγ antagonist (GW; GW9662) on transcription of genes involved in lipid droplet formation and ketone body utilization. Lower case letters and uppercase letters denote statistical differences (p < 0.05) between treatments without GW and treatments with GW, respectively. * denotes statistical difference (p < 0.05) between presence and absence of GW in the same LCFA treatment. For GW treatment alone, * denotes statistical difference (p < 0.05) with PCTR.