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. 2019 Jul 1;2019:9096409. doi: 10.1155/2019/9096409

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Copyright © 2019 Sirui Guo et al.

This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

PMC Copyright notice

KXS treatment inhibited oxidative stress and neuroinflammation-induced apoptosis in vitro. KXS treatment decreases the levels of ROS (a) and MMP (b), the concentration of IL-1β and TNF-α (c and d), and the expressions of Bax/bcl-2 and cleaved-caspase-3 (e and f), and the cell apoptosis rate (g and h) enhanced by Aβ _25-35 in PC12 cells. Values were the mean ± SD (n = 6). ^∗ P < 0.05, ^∗∗ P < 0.01, and ^∗∗∗ P < 0.001vs. the control group; ^# P < 0.05, ^## P < 0.01, and ^### P < 0.001vs. the Aβ group; and ^& P < 0.05, ^&& P < 0.01, and ^&&& P < 0.001vs. the LY+KXS group.