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. Author manuscript; available in PMC: 2020 Feb 15.
Published in final edited form as: Cancer. 2018 Nov 14;125(4):575–585. doi: 10.1002/cncr.31850

TABLE 3.

Associations Between Common Mutations, Pathways, and Treatment Center

Gene All
N = 81
No. (%)
Chile
N = 21
No. (%)
Japan
N = 11
No. (%)
United States
N = 49
No. (%)
Unadjusted Pa Adjusted Pa
Mutation burden (range)bb 5 (0−27) 7 (3−20) 6 (1−23) 4 (0−27) <.001 0.006
TP53 47 (58) 16 (76.2) 6 (54.5) 25 (51) .15 0.45
SMAD4 25 (30.9) 8 (38.1) 4 (36.4) 13 (26.5) .58 0.68
ARID1A 20 (24.7) 8 (38.1) 0(0) 12 (24.5) .045 0.35
ATM 15 (18.5) 3 (14.3) 2 (18.2) 10(20.4) .92 0.92
CDKN2A 12 (14.8) 4(19) 2 (18.2) 6 (12.2) .60 0.68
PIK3CA 10 (12.3) 3 (14.3) 0(0) 7(14.3) .56 0.68
TERT 10 (12.3) 3 (14.3) 2 (18.2) 5(10.2) .70 0.75
BRCA2 8 (9.9) 2 (9.5) 2 (18.2) 4 (8.2) .50 0.68
KMT2D 8 (9.9) 4(19) 1 (9.1) 3 (6.1) .21 0.45
ERBB3 7 (8.6) 0(0) 1 (9.1) 6 (12.2) .24 0.45
KRAS 7 (8.6) 4(19) 1 (9.1) 2 (4.1) .09 0.35
NF1 7 (8.6) 4(19) 1 (9.1) 2 (4.1) .09 0.35
AR 6 (7.4) 3 (14.3) 1 (9.1) 2 (4.1) .25 0.45
ARID2 6 (7.4) 0(0) 1 (9.1) 5 (10.2) .32 0.51
CDKN2B 6 (7.4) 3 (14.3) 1 (9.1) 2 (4.1) .25 0.45

All Chile Japan United States Unadjusted Adjusted

Pathway (n = 81) (n = 21) (n = 11) (n = 49) p-value p-value

Cell cycle 23 (28.4) 9 (42.9) 3 (27.3) 11 (22.4) .18 .62
Chromatin Remodeling 30 (37) 11 (52.4) 2 (18.2) 17 (34.7) .17 .62
NOTCH 20 (24.7) 8 (38.1) 2 (18.2) 10 (20.4) .28 .62
NRF2 6 (7.4) 1 (4.8) 1 (9.1) 4 (8.2) >.95 >.95
p53 59 (72.8) 18 (85.7) 8 (72.7) 33 (67.3) .31 .62
PIK3 26 (32.1) 9 (42.9) 2 (18.2) 15 (30.6) .40 .62
RTK-RAS 36 (44.4) 11 (52.4) 6 (54.5) 19 (38.8) .48 .62
TGF-β 31 (38.3) 10 (47.6) 4 (36.4) 17 (34.7) .64 .72
WNT 18 (22.2) 4(19) 4 (36.4) 10 (20.4) .44 .62

Abbreviations: AR, androgen receptor; ARID1A, AT-rich interaction domain 1A; ARID2, AT-rich interaction domain 2; ATM, ATM serine/threonine kinase; CDKN2A, cyclin-dependent kinase inhibitor 2A; CDKN2B, cyclin-dependent kinase inhibitor 2b; ERBB3, Erb-B2 receptor tyrosine kinase 3; NF1, neurofibromin 1; NRF2, nuclear factor (erythraid-derived 2)-like 2; PIK3, phosphatidylinositide 3-kinases; PIK3CA, phosphatidylinositol-4,5-bisphosphate 3-kinase catalytic subunit alpha; RTK-RAS, receptor tyrosine kinase-RAS, SMAD4, SMAD family member 4; TERT, telomerase reverse transcriptase; TGF-β, transforming growth factor β.

a

Bold type indicates statistical significance.

b

Mutation burden includes point mutations, copy number alterations (amplifications and deletions), and fusions.