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. 2019 Jul 11;10:1589. doi: 10.3389/fimmu.2019.01589

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Copyright © 2019 Demars, Lison, Machelart, Van Vyve, Potemberg, Vanderwinden, De Bolle, Letesson and Muraille.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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IFNγR deficiency leads to the massive recruitment of neutrophils in dermal tissue. Wild-type, IFNγR^−/−, IL-17RA^−/−, and TNFR1^−/− C57BL/6 mice were infected intradermally with a dose of 2 × 10⁴ CFU of mCherry-B. melitensis. Control wild-type mice were injected with PBS. Mice were sacrificed at 3 days post infection and the footpad was harvested and analyzed by fluorescent microscopy. The panels are color-coded with the text for DAPI, phalloidin, mCherry-B.melitensis, and LY-6G. Scale bar = 100 μm. These data are representative of two different experiments.