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. 2019 Jul 17;12:66. doi: 10.1186/s13041-019-0486-8

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — R18 provides potent neuroprotection against glutamic acid excitotoxicity in primary cortical neurons. Neuronal cultures were subjected to a 10-min R18 pre-treatment (2 μM) and subsequent 5-min glutamic acid exposure (Glut; 100 μM). MTS cell viability was assessed at 24 h post-injury. Cell viability was expressed as mean ± S.E.M (p < 0.05 relative to *Glut or #Cont)