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. 2019 Jul 16;13:718. doi: 10.3389/fnins.2019.00718

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Copyright © 2019 Mangin, Cogo, Moisan, Bonnin, Maïer and Kubis.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Assessment of long-term spatial memory between D30 and D38 in ND-PBS, untreated non-diabetic mice; ND-hADMSC, treated non-diabetic mice; D-PBS, untreated diabetic mice and D-hADMSC, treated diabetic mice (n = 9–16) (A) and NHT-PBS, untreated non-hypertensive mice; NHT-hADMSC, treated non-hypertensive mice; HT-PBS, untreated hypertensive mice and HT-hADMSC, treated hypertensive mice (n = 8–10) (B). hADMSC did not significantly modify the latency to escape of pMCAo mice whatever the risk factor; ^∗p < 0.05, ^∗∗p < 0.01, ^∗∗∗p < 0.001, ND-PBS versus D-PBS; ^#p < 0.05, ^##p < 0.01, ^###p < 0.001, ^####p < 0.0001 ND-PBS versus D-hADMSC; ^§p < 0.05, ^§§p < 0.01, ^§§§p < 0.001, ^§§§§p < 0.0001 ND-hADMSC versus D-PBS; ^$p < 0.05, ^$$p < 0.01, ^$$$p < 0.001, ^$$$$p < 0.0001 ND-hADMSC versus D-hADMSC.