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. 2018 Nov 6;56(7):4820–4837. doi: 10.1007/s12035-018-1401-5

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© The Author(s) 2018

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 6 — Prenatal BP-3 exposure modified the expression of neurogenesis-related and neurotransmitter receptor genes in mouse embryonic neurons. Gene expression patterns of neurogenesis demonstrated the 24 genes that were significantly differentially expressed between the control and BP-3-treated groups. Among these genes, 22 of them were upregulated (red color), and 2 genes were downregulated (green color) in the BP-3-treated samples compared to the control (a). Neurotransmitter receptor gene expression displayed 23 genes significantly differentially expressed between the control and BP-3-treated groups; 16 genes were upregulated (red color), and 7 genes were downregulated (green color) in the BP-3-treated samples compared to the controls (b)

Fig. 6 — Prenatal BP-3 exposure modified the expression of neurogenesis-related and neurotransmitter receptor genes in mouse embryonic neurons. Gene expression patterns of neurogenesis demonstrated the 24 genes that were significantly differentially expressed between the control and BP-3-treated groups. Among these genes, 22 of them were upregulated (red color), and 2 genes were downregulated (green color) in the BP-3-treated samples compared to the control (a). Neurotransmitter receptor gene expression displayed 23 genes significantly differentially expressed between the control and BP-3-treated groups; 16 genes were upregulated (red color), and 7 genes were downregulated (green color) in the BP-3-treated samples compared to the controls (b)