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. 2019 Mar 11;23(8):987–994. doi: 10.1007/s10157-019-01719-4

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 5 — The role of SIRT1 in podocytes under normal conditions. Under normal glucose conditions, retained SIRT1 deacetylates H3 and H4 histones, resulting in H3K9 histone methylation and Dnmt1 activation. H3K9 histone H3 Lys⁹, HMT histone methyltransferases, ME methyl-, DNMT DNA methyltransferases, CG CpG islands