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. 2019 Jul 16;30(7):855–864. doi: 10.1089/hum.2018.197

Figure 2.

Figure 2.

Biochemical correction of liver and heart following AAV vector and salmeterol administration. Acid α-glucosidase knockout (GAA-KO) mice were treated for AAV2/8-LSPhGAApA (AAV) either alone or in combination with salmeterol (Sal). Groups were as follows: untreated controls (GAA-KO mice: 4 males, 4 females); AAV only (GAA-KO mice: 5 males, 5 females); AAV+salmeterol (GAA-KO mice: 5 males, 5 females), and wildtype (WT; 5 males, 5 females). GAA activity and glycogen content for liver (A), heart (B), diaphragm (C), and quadriceps (D). Mean ± standard deviation is shown. *p < 0.05, **p < 0.01, ***p < 0.001, and ****p < 0.0001 for ANOVA with Dunnett test; AAV and untreated groups were used as controls.