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. 2019 Jun 17;6(7):1327–1331. doi: 10.1002/acn3.50802

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© Published 2019. This article is a U.S. Government work and is in the public domain in the USA. Annals of Clinical and Translational Neurology published by Wiley Periodicals, Inc on behalf of American Neurological Association.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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Parametric images of ¹¹C‐PBR28 total distribution volume corrected by plasma‐free fraction (V _T/f _P) in four patients with frontotemporal lobar degeneration (FTLD) (P1, P2, P3, and P4) and two representative healthy control subjects (bottom row). When visually compared to healthy control subjects, FTLD patients had greater ¹¹C‐PBR28 binding in frontotemporal cortices and other brain areas. Abbreviations: HAB, high‐affinity binder; MAB, mixed‐affinity binder; HC, healthy control.