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. 2019 Jul 23;9:10608. doi: 10.1038/s41598-019-46942-x

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Anti-NK1.1 mediated NK cell depletion abrogates the increased atherosclerosis observed in Cd47^−/− mice. WT and Cd47^−/− mice (n = 6–8) were injected once with AAV-PCSK9^DY and fed a HCD for 10 weeks and injected with anti-NK1.1 mAb for the last 7 weeks. Aortic sinus lesion area (A) and lesion area/vessel area percentage quantification (B). Plaque area stained with Oil Red O (C) and percentage of Oil Red O stained area (D).