Skip to main content
. 2019 Jul 24;2:270. doi: 10.1038/s42003-019-0503-6

Table 3.

Clinical features of patients with the SLC26A7 mutations and with the SLC26A4 mutations (Pendred syndrome)

SLC26A7 Pendred syndrome (SLC26A4)
Our study Previous report
Patient 1 Patient 2 Elder sister (43.1) Younger sister (43.2)
Mutation c.1498C > T c.927_933delinsG
Zygosity Homozygous Homozygous
Amino acid change p.Gln500Ter p.Ile309_Glu311delinsMet
Thyroid
  Hypothyroidism* Yes Yes Yes Yes Euthyroid in most patients
  Goitre Yes Yes Yes No 50–83% of the patients
  Severity of hypothyroidism Moderate Severe N.A. N.A. N.A.
  Age at diagnosis of hypothyroidism 15 days 5 years 3 years N.A.
  High level of serum TG Yes Yes N.A. N.A. Yes
Other Feature
  Renal tubular acidosis* No No No No N.A.
  Deafness No No N.A. N.A. Yes
  EVA N.A. N.A. N.A. N.A. 80–100% of the patients
  Impairment of gastric acid secretion* N.A. N.A. N.A. N.A. N.A.

TG, thyroglobulin; EVA, enlarged vertibular aqueduct; N.A., not available

*Phenotype of Slc26a7 knock out mice