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. 2019 Jul 25;8(8):355–367. doi: 10.1089/wound.2018.0931

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© Sandeep Dhall, et al. 2019; Published by Mary Ann Liebert, Inc.

This Open Acess article is distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — VLAM regenerates skin tissue in a diabetic mouse wound model. The integrity of skin tissue post-wound closure in control, DLAM- and VLAM-treated animals was assessed histologically. Wound tissue post-closure was collected and stained with H&E (a–c) or MT (d–f). Skin structures in the control and DLAM-treated wound lack dermal-epidermal junctions (c, black arrows). Complete regeneration of skin tissue was observed in a VLAM-treated wound (b, d). H&E, hematoxylin and eosin; MT, Masson's trichrome.