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. Author manuscript; available in PMC: 2019 Jul 29.
Published in final edited form as: Alcohol Clin Exp Res. 2016 May 14;40(7):1454–1461. doi: 10.1111/acer.13097

Fig. 1.

Fig. 1.

Effects of IP3 inhibitor6 xestospongin C (0.5 lM) application on NeuN/Fox-3 immunofluorescence in ethanol (EtOH)-exposed and EtOHna€ıve hippocampi within the pyramidal cell layer of the CA1 (A), CA3 (B), and the granule cell layer of the dentate gyrus (DG) (C). Data are presented as percent control of the mean SEM. *Statistical significance (p < 0.05) compared to EtOH- and -drug-na€ıve hippocampi; #statistical significance (p < 0.05) compared to EtOH-treated, but drug-na€ıve hippocampi. N = 27 for EtOH-treated hippocampi; N = 29 for hippocampi co-exposed to EtOH and xestospongin C; N = 33 for EtOH- and drug-na€ıve hippocampi; N = 30 for EtOH-na€ıve hippocampi exposed to xestospongin C.