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. Author manuscript; available in PMC: 2019 Jul 29.
Published in final edited form as: Alcohol. 2017 Aug 30;64:37–43. doi: 10.1016/j.alcohol.2017.05.006

Fig. 3.

Fig. 3.

Neither ethanol-treated hippocampi nor control-treated hippocampi were significantly altered following application of protein kinase inhibitor KT-5720 (1 mM) in the pyramidal cell layer of CA3. Data are presented as percent control of the mean ± SEM. **Statistical significance (p < 0.05) compared to control-treated hippocampi. N ¼ 40 for ethanol-treated hippocampi; N ¼ 40 for hippocampi treated with ethanol and KT during EWD; N ¼ 40 for hippocampi treated with ethanol and KT concomitantly; N ¼ 38 for control-treated hippocampi; N ¼ 36 for control-treated hippocampi with KT applied during the EWD period; N ¼ 25 for control-treated hippocampi with KT applied during CIE.