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. 2019 Jul 23;13:767. doi: 10.3389/fnins.2019.00767

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Copyright © 2019 Durán-Laforet, Fernández-López, García-Culebras, González-Hijón, Moraga, Palma-Tortosa, García-Yébenes, Vega-Pérez, Lizasoain and Moro.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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(A) Representative images of the whole area of study comprising the healthy tissue, the peri-infarct and the infarct. (B,C) Representative confocal fluorescence micrographs (20x) showing perfusion intensity in ischemic and contralateral regions at 72 h (B) and 5 days (C) after pMCAO or 60-min tMCAO. Intravascular perfusion (lectin, gray) was analyzed in Glut-1⁺ vessels (green). (D) Quantification of average lectin intensity in vessels expressed as ratio of ischemic/contralateral (internal control) sub-acutely (n = 6–7). ^* Student’s t-test p < 0.05 vs. pMCAO.