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. 2019 Jul 29;17:86. doi: 10.1186/s12964-019-0393-8

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Effect of HDAC1 knockdown on doxorubicin resistance of HL-60/ADM, K562/A02, and primary BMCs of refractory AML. a Relative cell viability of si-HDAC1 transfected AML cells after different doses of doxorubicin treatment. b Percentage of apoptotic cells in si-HDAC1 transfected AML cells after doxorubicin (0.01 μg/ml) treatment. c doxorubicin-releasing index of si-HDAC1 transfected AML cells. d MRP1 protein expression in si-HDAC1 transfected AML cells. DOX, doxorubicin. Data are the mean ± SD of three independent experiments in AML cells. ^*P < 0.05, ^**P < 0.01 vs. si-control